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The primary mechanism involves tyrosinase inhibition, where glutathione binds to copper at the enzyme’s active sites to reduce melanin synthesis

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T-bet, a transcription factor, is instrumental in regulating the balance between Th1 and Th2 cell populations (132), and it has been noted that tumor progression, particularly in melanoma, is frequently linked to a shift from Th1 to Th2 dominance (133) (134)